Study guide

Dental Charting & Hard Tissue Assessment

ADEX Dental Hygiene Clinical Examination Exam

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1 Introduction & Clinical Rules

About the ADEX Dental Hygiene Exam SPTCE Clinical Rules & Patient Selection

2 Medical & Dental Assessment

Medical History & Vital Signs Pharmacological Risks & Contraindications Systemic Red Flags & Informed Consent

3 Intra & Extra Oral Assessment & Charting

Extra-oral & Intra-oral Soft Tissue Exam Dental Charting & Hard Tissue Assessment Periodontal Probing & Charting 2017 AAP Periodontal Classification

4 Radiology & Imaging

Radiographic Prescription, Exposure & Safety Radiographic Interpretation of Pathologies & Structures Radiographic Interpretation of Periodontal Disease

5 Dental Hygiene Care Planning

Diagnosis & Care Plan Prioritization Recall Intervals & Patient Self-Care Education Re-evaluation & Treatment Outcomes

6 Patient Care & Instrumentation

Hand Instrumentation & Ergonomics Powered Instrumentation & Preventive Agents Local Anesthesia & Pain Control Medical Emergency Protocols

7 Dental Specialties & Applied Pharmacology

Dental Specialties & Patient Management Applied Pharmacology

Dental Charting & Hard Tissue Assessment

Key Takeaways Explain This Topic AI

G.V. Black's classification system categorizes caries and restorations into 6 distinct classes based on the involved tooth surfaces.

Dental restorations are radiographically distinct, ranging from the 100% radiopacity of amalgam/gold to the moderate 50-70% radiopacity of composite resins.

Amelogenesis imperfecta is a hereditary defect (1 in 14,000) affecting enamel, whereas dentinogenesis imperfecta (1 in 8,000) results in opalescent crowns and obliterated pulps.

Peg laterals are microdontia defects affecting the maxillary lateral incisors (teeth #7 and #10), occurring in approximately 1.5% of the population.

Dental implants are titanium fixtures requiring 3 to 6 months of healing to achieve structural osseointegration with alveolar bone.

Last updated: July 2026

  • Dental Charting & Hard Tissue Assessment
  • Documentation of Dental Restorations

Accurate charting of existing dental restorations is critical for treatment planning, forensic identification, and monitoring material integrity over time. Restorative materials are identified through visual inspection, tactile evaluation with an explorer, and radiographic interpretation.

1. Dental Amalgam

Clinical Appearance: Silver-gray metallic appearance, turning dark gray or black as it tarnishes.

Radiographic Appearance: Radiopaque (solid white) with irregular, distinct borders. It is denser than composite and lacks the precise, smooth margins of cast metal restorations.

2. Composite Resin

Clinical Appearance: Tooth-colored material. Older composites may appear stained or rough, while modern formulations blend seamlessly with natural tooth structure.

Radiographic Appearance: Varies from radiolucent to moderately radiopaque depending on the filler content. It is distinguished from caries by its well-defined, smooth cavity preparation outlines.

3. Crowns and Fixed Prosthodontics

Full Gold Crown (FGC): Smooth, highly polished metallic margins. Radiographically, it appears completely radiopaque with smooth contours that cover the entire clinical crown.

Porcelain-Fused-to-Metal (PFM): Combines the esthetics of porcelain with the strength of a metal substructure. Radiographically, the metal framework appears completely radiopaque, outlined by a thin, moderately radiopaque layer of porcelain.

All-Ceramic (Zirconia/Lithium Disilicate): Tooth-colored. Radiographically, they appear semi-radiopaque (zirconia is highly radiopaque, while lithium disilicate is less dense).

Bridge (Fixed Partial Denture): Replaces one or more missing teeth. It consists of abutments (the supporting teeth restored with crowns) and pontics (the suspended artificial teeth).

Dental Implant: A titanium post surgically placed into the alveolar bone to replace a missing root. Radiographically, it appears as a highly radiopaque, screw-like fixture. It must achieve osseointegration-a direct bone-to-implant contact without intervening soft tissue.

G.V. Black's Classification of Caries and Restorations

Dr. G.V. Black developed a standardized system for classifying carious lesions and restorations based on their anatomical location. This system remains the universal standard for dental charting:

Class Anatomical Description Common Locations

Class I Pits and fissures of posterior and anterior teeth Occlusal of molars/premolars; facial/lingual pits of molars; lingual pits of maxillary incisors

Class II Proximal surfaces of posterior teeth Mesial or distal surfaces of molars and premolars

Class III Proximal surfaces of anterior teeth, not involving the incisal angle Mesial or distal surfaces of incisors and canines

Class IV Proximal surfaces of anterior teeth, involving the incisal angle Mesial-incisal or distal-incisal surfaces of incisors and canines

Class V Gingival third of the facial or lingual surfaces of any tooth Cervical region of anterior or posterior teeth (not pit/fissure)

Class VI Cusp tips of posterior teeth or incisal edges of anterior teeth Tips of cusps on molars/premolars; incisal edges of central/lateral incisors

Developmental Anomalies of the Hard Tissues

Developmental disturbances during odontogenesis can alter the structure, color, and morphology of the enamel, dentin, or entire tooth crown.

1. Amelogenesis Imperfecta (AI)

Amelogenesis imperfecta is a hereditary ectodermal disorder that interferes with the deposition, calcification, or maturation of enamel matrix.

Clinical Findings: Enamel is thin, soft, pitted, or grooved. It ranges in color from chalky white to yellow-brown. The enamel is poorly bonded to the dentin and easily chips or wears away, exposing the underlying dentin to rapid attrition.

Radiographic Findings: The enamel is extremely thin or entirely absent. When present, its radiodensity is significantly reduced, often appearing equivalent to the underlying dentin, which obliterates the normal enamel-dentin contrast.

2. Dentinogenesis Imperfecta (DI)

Dentinogenesis imperfecta is an autosomal dominant mesodermal disorder affecting dentin formation.

Clinical Findings: Teeth exhibit a characteristic translucent, opalescent, gray-to-bluish-brown coloration. Although the enamel is chemically normal, it fractures off rapidly because the underlying dentin is abnormally soft and provides poor physical support.

Radiographic Findings: Teeth exhibit bulbous, bell-shaped crowns with a marked constriction at the cementoenamel junction (CEJ), often referred to as a 'tulip-like' appearance. The roots are short, thin, and tapered. A hallmark sign is the early, complete obliteration of the pulp chambers and root canals due to rapid, unregulated dentin deposition.

3. Dental Fluorosis

Fluorosis is a form of enamel hypomineralization caused by the chronic ingestion of excessive fluoride during amelogenesis (specifically before age 8, when crowns are forming).

Clinical Findings: Ranges from mild (opaque, white flecks or paper-white chalky lines) to moderate (affecting over 50% of the tooth surface) to severe (widespread brown staining, pitting, and structural brittleness). Teeth with fluorosis are highly resistant to caries due to the fluorapatite structure.

4. Microdontia and Peg Laterals

Microdontia: A condition where one or more teeth are physically smaller than normal. Can be generalized (associated with endocrine disorders) or localized.

Peg Laterals: The most common form of localized microdontia. It affects the maxillary lateral incisors, resulting in a small, conical, or peg-shaped crown. They are hereditary and often occur bilaterally.

Charting Missing and Impacted Teeth

Congenitally Missing (Hypodontia): The developmental absence of one or more teeth. The most commonly affected teeth are the third molars, followed by the mandibular second premolars and the maxillary lateral incisors. Clinically, a missing tooth space is noted, and radiographs are required to confirm the absence of a developing tooth bud.

Impacted Teeth: Teeth that remain unerupted within the alveolar bone beyond their normal eruption age due to a physical barrier (such as bone, overlying fibrous tissue, or an adjacent tooth root). Maxillary and mandibular third molars are the most commonly impacted teeth, followed closely by maxillary canines.