10.4 Submersion Injuries, Drowning & Thermal/Burn Emergencies

BCEN CPEN Certified Pediatric Emergency Nurse Exam

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1 Chapter 1: Exam Overview, Triage Process & Initial Emergency Assessment

1.1 CPEN Exam Logistics, Blueprint & Certification Overview 1.2 Pediatric Triage Systems & Acuity Categorization 1.3 Pediatric Assessment Triangle (PAT) & Initial Impression 1.4 Primary & Secondary Pediatric Emergency Assessment 1.5 Age-Specific Vital Signs, Developmental Considerations & Pain Scales

2 Chapter 2: Pediatric Respiratory Emergencies

2.1 Upper Airway Obstruction 2.2 Lower Airway Emergencies 2.3 Respiratory Distress vs. Respiratory Failure & Airway Management

3 Chapter 3: Pediatric Cardiovascular Emergencies

3.1 Pediatric Dysrhythmias & ECG Analysis 3.2 Congenital Heart Defects 3.3 Acquired Heart Disease & Heart Failure

4 Chapter 4: Pediatric Neurological Emergencies

4.1 Pediatric Seizures, Status Epilepticus & Febrile Seizures 4.2 Altered Mental Status, Increased ICP & Head Trauma 4.3 Pediatric Central Nervous System Infections

5 Chapter 5: Pediatric Gastrointestinal & Genitourinary Emergencies

5.1 Pediatric GI Surgical Emergencies 5.2 Pediatric Dehydration, Gastroenteritis & Fluid Resuscitation Protocols 5.3 Pediatric GU & Renal Emergencies

6 Chapter 6: Pediatric Endocrine, Metabolic & Hematologic/Oncologic Emergencies

6.1 Diabetic Ketoacidosis (DKA) & Hypoglycemia in Pediatric Emergency Patients 6.2 Electrolyte Imbalances & Inborn Errors of Metabolism 6.3 Sickle Cell Disease Crisis & Pediatric Oncologic Emergencies

7 Chapter 7: Pediatric Musculoskeletal, Integumentary & ENT/Ophthalmic Emergencies

7.1 Pediatric Musculoskeletal Injuries & Fractures 7.2 Pediatric Dermatologic Emergencies & Rashes 7.3 Pediatric ENT & Ocular Emergencies

8 Chapter 8: Pediatric Trauma Emergencies

8.1 Pediatric Trauma Resuscitation & Anatomical Vulnerabilities 8.2 Head, Spinal & Thoracic Trauma in Children 8.3 Abdominal, Pelvic & Extremity Trauma in Pediatric Patients

9 Chapter 9: Child Maltreatment, Toxicology & Behavioral Emergencies

9.1 Child Abuse, Neglect, Non-Accidental Trauma & Mandatory Reporting 9.2 Pediatric Toxicology, Toxidromes & Emergency Antidotes 9.3 Pediatric Behavioral & Mental Health Crisis Management

10 Chapter 10: Pediatric Multi-System Emergencies, Shock & Thermal Injuries

10.1 Pediatric Shock Recognition & Classification 10.2 Pediatric Sepsis, Septic Shock & Multiple Organ Dysfunction Syndrome 10.3 Pediatric Anaphylaxis & Severe Allergic Reactions 10.4 Submersion Injuries, Drowning & Thermal/Burn Emergencies

11 Chapter 11: Professional Practice, Legal, Ethical & Safety Standards

11.1 Legal & Ethical Considerations 11.2 Patient Safety, Medication Error Prevention & Weight-Based Dosing 11.3 Family-Centered Care, Communication & Disaster/Surge Preparedness

10.4 Submersion Injuries, Drowning & Thermal/Burn Emergencies

Key Takeaways Explain This Topic AI

Drowning is primary respiratory impairment from submersion/immersion in liquid leading to laryngospasm, surfactant inactivation, non-cardiogenic pulmonary edema (ARDS), severe hypoxia, and hypoxic-ischemic brain injury.

Resuscitation from severe hypothermia requires maintaining CPR until the patient is rewarmed to >32°C–35°C ('not dead until warm and dead'); limit defibrillation to 1–3 shocks and hold IV ACLS medications when core body temperature is <30°C.

Pediatric burn TBSA assessment must utilize the Lund-Browder chart rather than the adult Rule of Nines because children have a disproportionately larger head surface area (18% in infants) and smaller lower extremity surface area (14% per leg in infants).

Burn fluid resuscitation uses the Modified Parkland formula (3–4 mL Lactated Ringer's × kg × % TBSA); for pediatric patients <30 kg, maintenance fluids containing Dextrose (e.g. D5W with 0.45% NS) MUST be added concurrently to prevent severe hypoglycemia.

Inhalation injury is characterized by facial burns, soot in the oropharynx, carbonaceous sputum, and stridor; early elective endotracheal intubation is mandatory before progressive airway swelling causes complete laryngeal obstruction.

Last updated: July 2026

10.4 Submersion Injuries, Drowning & Thermal/Burn Emergencies

Submersion injuries, severe hypothermia, and thermal burn trauma represent multi-system pediatric emergencies requiring precise, protocolized nursing care. Pediatric anatomy—specifically a large surface-area-to-body-mass ratio, thinner skin, smaller glycogen stores, and non-compliant pediatric airways—amplifies the physiological impact of environmental trauma.

Pathophysiology of Drowning & Submersion Injuries

Drowning is defined as the process resulting in primary respiratory impairment from submersion or immersion in liquid. (Outdated terms such as 'near-drowning', 'wet drowning', or 'dry drowning' must no longer be used). +-----------------------------------------------------------------------------------------+

• | PATHOPHYSIOLOGY OF DROWNING |
• +-----------------------------------------------------------------------------------------+
• | SUBMERSION IN LIQUID -> Involuntary breath-holding & liquid swallowing |
• | | |
• | v |
• | REFLEX LARYNGOSPASM -> Inability to ventilate -> Hypoxia & Hypercapnia |
• | | |
• | v |
• | LIQUID ASPIRATION -> Washout & deactivation of pulmonary surfactant |
• | | |
• | v |
• | ALVEOLAR COLLAPSE & ATELECTASIS -> Non-cardiogenic Pulmonary Edema (ARDS) |
• | | |
• | v |
• | PROFOUND HYPOXEMIA & ISCHEMIA -> Hypoxic-Ischemic Encephalopathy (HIE) & Cardiac Arrest|
• +-----------------------------------------------------------------------------------------+
• Clinical Management Priorities

Hypoxia is the Primary Instigator : Resuscitation begins with immediate ventilation (5 initial rescue breaths) followed by conventional CPR (30:2 single rescuer, 15:2 two-rescuer).

Pulmonary Complications : Aspiration of fresh or salt water damages alveolar-capillary membranes and washes out surfactant, leading to intrapulmonary shunting, non-cardiogenic pulmonary edema, and Acute Respiratory Distress Syndrome (ARDS) .

Cervical Spine Immobilization : Indicated ONLY if there is a history of diving, high-speed watercraft trauma, falling from height, or signs of blunted trauma.

Observation Threshold : Any pediatric submersion patient who experienced loss of consciousness, required resuscitation, or displays tachypnea, cough, or crackles must be monitored for at least 6 to 8 hours , as ARDS can develop insidiously.

Hypothermia Resuscitation Protocols

Hypothermia occurs rapidly in submerged or burned children due to high body surface area. Core body temperature classification:

Mild Hypothermia : 32 ∘ C − 35 ∘ C 32^\circ C - 35^\circ C 3 2 ∘ C − 3 5 ∘ C ( 89.6 ∘ F − 95 ∘ F 89.6^\circ F - 95^\circ F 89. 6 ∘ F − 9 5 ∘ F ). Passive external rewarming.

Moderate Hypothermia : 28 ∘ C − 32 ∘ C 28^\circ C - 32^\circ C 2 8 ∘ C − 3 2 ∘ C ( 82.4 ∘ F − 89.6 ∘ F 82.4^\circ F - 89.6^\circ F 82. 4 ∘ F − 89. 6 ∘ F ). Active external rewarming.

Severe Hypothermia : < 28 ∘ C < 28^\circ C < 2 8 ∘ C ( < 82.4 ∘ F < 82.4^\circ F < 82. 4 ∘ F ). Active internal (core) rewarming. +-----------------------------------------------------------------------------------------+

• | HYPOTHERMIA RESUSCITATION MODIFICATIONS |
• +-----------------------------------------------------------------------------------------+
• | CORE TEMP < 30°C (86°F) | - Limit defibrillation shocks to 1 - 3 attempts |
• | | - WITHHOLD ALL IV ACLS MEDICATIONS (Epineph/Amio) |
• | | - Initiate aggressive active core rewarming |
• +--------------------------+--------------------------------------------------------------+
• | CORE TEMP 30°C - 35°C | - Double the time interval between IV ACLS medication doses |
• | | - Continue active external and internal rewarming |
• +--------------------------+--------------------------------------------------------------+
• | CPR TERMINATION RULE | - "PATIENT IS NOT DEAD UNTIL WARM AND DEAD" |
• | | - Continue CPR until core temp > 32°C - 35°C before stopping |
• +-----------------------------------------------------------------------------------------+
• Rewarming Methods

Passive External : Remove wet clothing, warm dry blankets, warm ambient room ( 30 ∘ C 30^\circ C 3 0 ∘ C ).

Active External : Radiant warmers, forced-air warming blankets (Bair Hugger), warm water immersion.

Active Internal (Core) : Humidified warm oxygen ( 42 ∘ C − 46 ∘ C 42^\circ C - 46^\circ C 4 2 ∘ C − 4 6 ∘ C ), warm IV fluids ( 39 ∘ C − 42 ∘ C 39^\circ C - 42^\circ C 3 9 ∘ C − 4 2 ∘ C ), warm pleural or peritoneal lavage, extracorporeal membrane oxygenation (ECMO) / cardiopulmonary bypass.

Pediatric Burn Assessment: Lund-Browder vs. Rule of Nines

Accurate calculation of Total Body Surface Area (TBSA) burned is essential for fluid resuscitation. Superficial (1st-degree) burns (sunburns) ARE EXCLUDED from TBSA fluid calculations. Only partial-thickness (2nd-degree) and full-thickness (3rd/4th-degree) burns are included.

Why the Adult Rule of Nines Fails in Children

Infants and young children have a significantly larger head surface area and smaller lower extremity surface area relative to adults. Using the adult Rule of Nines severely underestimates head burn TBSA and overestimates leg burn TBSA in pediatric patients. +-----------------------------------------------------------------------------------------+

• | BURN TBSA ESTIMATION: LUND-BROWDER vs. RULE OF NINES |
• +-----------------------------------------------------------------------------------------+
• | BODY REGION | INFANT (< 1 YEAR) | 5-YEAR-OLD CHILD | ADULT |
• +------------------------+-----------------------+--------------------+-------------------+
• | Head & Neck | **18%** | **13%** | 9% |
• | Anterior Trunk | 18% | 18% | 18% |
• | Posterior Trunk | 18% | 18% | 18% |
• | Each Upper Extremity | 9% | 9% | 9% |
• | Each Lower Extremity | **14%** | **16.5%** | 18% |
• | Perineum | 1% | 1% | 1% |
• +-----------------------------------------------------------------------------------------+

Palmar Method for Patchy Burns : The area of the patient's hand (palm plus digits) equals approximately 1% TBSA . Use this method to quickly estimate scattered burn patches.

Pediatric Burn Fluid Resuscitation Formulas

Massive capillary leak occurs following burn trauma, peaking at 8 to 24 hours post-injury.

1. Modified Parkland Resuscitation Formula Total 24-Hour Resuscitation Volume = ( 3 to 4 mL ) × Weight (kg) × % TBSA (2nd and 3rd degree burns) \text{Total 24-Hour Resuscitation Volume} = (3 \text{ to } 4 \text{ mL}) \times \text{Weight (kg)} \times \% \text{TBSA (2nd and 3rd degree burns)} Total 24-Hour Resuscitation Volume = ( 3 to 4 mL ) × Weight (kg) × % TBSA (2nd and 3rd degree burns)

Fluid of Choice : Lactated Ringer's (LR).

Time Administration Schedule :

First 50% of total calculated volume is administered over the first 8 hours post-injury ( calculated from the time the burn occurred, NOT ED arrival time ).

Remaining 50% of volume is administered over the next 16 hours .

2. CRITICAL MANDATE: Concurrent Dextrose Maintenance

Young children ( < 30 kg < 30 \text{ kg} < 30 kg ) have limited hepatic glycogen reserves. If resuscitated solely with dextrose-free Parkland fluid, they will develop life-threatening hypoglycemia .

THE CPEN BURN RULE : For children < 30 kg < 30 \text{ kg} < 30 kg , emergency nurses MUST administer maintenance fluids containing Dextrose (e.g., D 5 0.45 % Normal Saline D_5 0.45\% \text{ Normal Saline} D 5 ​ 0.45% Normal Saline ) CONCURRENTLY with Parkland LR fluid resuscitation! 4-2-1 Rule for Hourly Maintenance = ( 4 mL/kg for first 10 kg ) + ( 2 mL/kg for next 10 kg ) + ( 1 mL/kg for remaining kg ) \text{4-2-1 Rule for Hourly Maintenance} = (4 \text{ mL/kg for first 10 kg}) + (2 \text{ mL/kg for next 10 kg}) + (1 \text{ mL/kg for remaining kg}) 4-2-1 Rule for Hourly Maintenance = ( 4 mL/kg for first 10 kg ) + ( 2 mL/kg for next 10 kg ) + ( 1 mL/kg for remaining kg )

Resuscitation Target: Urine Output Titration

Do not titrate burn fluids based on blood pressure alone! Titrate fluid rates to maintain goal urine output :

Infants & Children (< 30 kg) : 1.0 − 2.0 mL/kg/hr 1.0 - 2.0 \text{ mL/kg/hr} 1.0 − 2.0 mL/kg/hr .

Older Children & Adolescents (> 30 kg) : 0.5 − 1.0 mL/kg/hr 0.5 - 1.0 \text{ mL/kg/hr} 0.5 − 1.0 mL/kg/hr .

Inhalation Injury & Thermal Airway Emergencies

Inhalation injury dramatically increases mortality in burn trauma. +-----------------------------------------------------------------------------------------+

• | INHALATION INJURY CLINICAL TRIAD |
• +-----------------------------------------------------------------------------------------+
• | PHYSICAL SIGNS | Facial burns, singed nasal hairs, soot in mouth, dark sputum |
• | AIRWAY SYMPTOMS | Hoarseness, brassy cough, inspiratory stridor, intercostal retractions|
• | SYSTEMIC TOXICITY | Carbon Monoxide (CO) poisoning & Cyanide toxicity |
• +-----------------------------------------------------------------------------------------+
• 1. Thermal Airway Edema

Direct heat causes rapid supraglottic edema. Immediate elective endotracheal intubation is mandatory at the earliest sign of airway involvement (hoarseness, stridor, soot in pharynx) before thermal edema completely occludes the glottic opening.

2. Carbon Monoxide (CO) Poisoning

CO binds to hemoglobin with an affinity 200 times greater than oxygen, forming carboxyhemoglobin (COHb) and shifting the oxyhemoglobin dissociation curve to the left.

Pulse Oximetry Trap : Standard pulse oximeters ( S p O 2 SpO_2 Sp O 2 ​ ) cannot distinguish carboxyhemoglobin from oxyhemoglobin and will falsely report 100% saturation !

Diagnosis : Co-oximetry arterial or venous blood gas measuring C O H b COHb CO H b percentage.

Treatment : Administer 100 % 100\% 100% high-flow oxygen via non-rebreather mask (reduces COHb half-life from 320 minutes to 80 minutes) or hyperbaric oxygen for C O H b > 25 % COHb > 25\% CO H b > 25% or neurological compromise.

3. Cyanide Toxicity

Produced by combustion of synthetic polymers and plastics. Suspect in patients with severe lactic acidosis ( > 8 − 10 mmol/L > 8 - 10 \text{ mmol/L} > 8 − 10 mmol/L ) out of proportion to burn size.

Antidote : Intravenous Hydroxocobalamin (Cyanokit) 70 mg/kg 70 \text{ mg/kg} 70 mg/kg IV (max 5 g 5 \text{ g} 5 g ), which binds cyanide to form non-toxic vitamin B 12 a B_{12a} B 12 a ​ (cyanocobalamin) excreted in urine.